Why Do 2-Year-Olds Have Higher Cholesterol Levels and What Can Be Done About It

Typically, unless a 2-year-old has a rare genetic disorder with a strong family history, their cholesterol levels will be very normal. However, autopsy research on children who died of other conditions, such as motor vehicle accidents, shows the start of atherosclerosis plaques and artery hardening as young as 2 years old. This condition is alarming because it suggests that even in early childhood, the body begins to accumulate harmful health conditions.

The Early Onset of Atherosclerosis

During autopsies, researchers have found the earliest signs of atherosclerosis in children as young as 2 years old. This premature development of arteries lined with plaques is concerning for several reasons. It indicates that even at a young age, there are underlying mechanisms at play that contribute to chronic diseases. These findings emphasize the importance of early intervention and preventive measures to maintain cardiovascular health.

The Initial Treatment for Children and Adults

The initial treatment for atherosclerosis, whether in children or adults, is remarkably similar. A plant-based diet, regular exercise for 60 minutes a day, stress reduction, and adequate sleep are all essential components of the treatment plan. Adhering to these guidelines can help manage and prevent the progression of atherosclerosis, leading to a healthier lifestyle overall.

For those seeking more comprehensive solutions, there are detailed guides and tips available online. By following these strategies, individuals can potentially clean out plaque buildup from their arteries, lower cholesterol levels to a healthy range, and enhance both physical and mental energy to levels that were previously not thought possible. Some remarkable success stories have even seen their overall cholesterol decrease by 60 points, their triglycerides by 51 points, and LDL by 37 points!

The Role of Nature and Nurture

The reasons behind the premature onset of atherosclerosis in children are multifaceted, encompassing both genetic and environmental factors. Recent research in epigenetics sheds light on how our diet can affect gene expression. Specific dietary components can lead to the methylation of DNA, turning certain genes on or off. The Standard American Diet (SAD) seems to promote the activation of genes associated with high cholesterol, heart disease, diabetes, and early death, not only in the current generation but potentially affecting future generations as well.

This genetic predisposition is amplified by environmental factors, such as poor dietary choices, lack of exercise, chronic stress, and inadequate sleep. Together, these factors create a perfect storm for the development of atherosclerosis in young children. By making lifestyle changes, we can actively work against these negative influences and promote better health outcomes.

Conclusion

While a rare genetic disorder is the primary factor for higher cholesterol levels in very young children, the majority of cases can be linked to lifestyle and environmental factors. By focusing on a balanced diet, regular exercise, managing stress, and ensuring adequate sleep, we can mitigate the risk of atherosclerosis and promote better overall health. With the right strategies and determination, we can safeguard our children's health and potentially reverse the harmful processes that could lead to chronic conditions later in life.